(訪談)自體免疫病,最常見是橋本症(甲狀腺發炎),自然療法,柏格醫生 Dr Berg
Summary
TLDR本视频访谈了经验丰富的医生Dr. Dimitri,探讨了自身免疫性疾病的流行情况及其对健康的深远影响。Dr. Dimitri指出,自身免疫疾病已成为美国慢性疾病的主要原因,患者数量超过心血管疾病和癌症患者总和。特别地,他强调了桥本甲状腺炎(Hashimoto's disease)作为最常见的自身免疫性疾病,导致甲状腺功能减退。视频中讨论了该疾病的诊断、潜在原因、症状以及可能的逆转方法,同时强调了营养、代谢功能和微生物组在疾病发展中的作用。
Takeaways
- 📊 自身免疫疾病已成为导致慢性疾病的主要原因,在美国有5000万人患有某种自身免疫疾病,超过心血管疾病和癌症患者人数的总和。
- 🏥 哈希莫托病(Hashimoto's disease)是最常见的自身免疫疾病,是导致甲状腺功能减退(hypothyroidism)的主要原因。
- 👩⚕️ Dr. Dimitri 强调,自身免疫疾病不仅仅是器官疾病,更是免疫系统攻击自身组织的慢性炎症性疾病。
- 🔬 哈希莫托病的诊断通常不通过活检,而是通过血液检测甲状腺激素水平(TSH、T3、T4)和超声波检查甲状腺结构。
- ⏳ 自身免疫疾病的治疗可能需要较长时间,甲状腺细胞的再生周期为七年,这使得疾病逆转变得复杂。
- 🛑 哈希莫托病的治疗目标首先是降低自身抗体水平,停止免疫系统对甲状腺的破坏,其次是控制炎症,减少对甲状腺功能的进一步损害。
- 🌡️ 甲状腺功能减退的常见症状包括能量水平下降、体重调节困难、激素失衡、情绪不稳定等。
- 💊 哈希莫托病患者普遍存在维生素D缺乏,以及胰岛素抵抗或代谢功能障碍。
- 🥗 患者还可能存在多种微量营养素的边际缺乏,这些缺乏会影响线粒体功能,降低能量水平和身体效率。
- 🧬 Dr. Dimitri 使用代谢组学测试来评估患者体内的代谢物,以了解营养素的实际利用情况,从而提供个性化的治疗建议。
- 🔗 通过测量代谢物之间的化学转化,可以更准确地评估身体对特定营养素的需求,而不是仅仅依赖血液浓度。
Q & A
为什么自身免疫性疾病比心血管疾病和癌症更为常见?
-根据Dr. Demitri的统计数据,在美国,患有自身免疫性疾病的人数约为5000万,这远超过癌症患者(900万)和心血管疾病患者(2200万)的总和。
什么是自身免疫性疾病?
-自身免疫性疾病是指免疫系统无法识别自身的组织或器官,将其视为外来物质并进行攻击,导致慢性炎症和器官损伤。
最常见的自身免疫性疾病是什么?
-最常见的自身免疫性疾病是桥本甲状腺炎,这是一种自身免疫性甲状腺疾病。
桥本甲状腺炎的主要症状是什么?
-桥本甲状腺炎的主要症状包括低能量、体重调节困难、荷尔蒙失衡和情绪不稳定等。
为什么桥本甲状腺炎患者大多数是女性?
-大多数桥本甲状腺炎患者是女性,因为女性在一生中经历了许多荷尔蒙变化,如月经、怀孕和更年期,这些变化会导致免疫系统适应性失调。
桥本甲状腺炎是否可逆转?
-桥本甲状腺炎在早期阶段可以通过降低自身抗体和控制炎症来停止甲状腺破坏,从而部分逆转病情。然而,对于已经开始服用甲状腺激素药物的患者,完全停止药物治疗的可能性较小。
哪些因素可能引发桥本甲状腺炎?
-引发桥本甲状腺炎的因素包括荷尔蒙变化、维生素D缺乏、胰岛素抵抗和微量营养素缺乏等。
如何诊断桥本甲状腺炎?
-桥本甲状腺炎的诊断通常通过测量TSH、T3和T4激素水平,以及超声检查甲状腺结构来进行。最终确诊通常需要进行活检。
为什么维生素D缺乏在桥本甲状腺炎患者中如此普遍?
-所有桥本甲状腺炎患者都存在维生素D缺乏,这可能与维生素D在免疫系统调节中的关键作用有关。
如何通过营养调整来改善桥本甲状腺炎?
-通过补充维生素D、改善胰岛素敏感性和纠正微量营养素缺乏,可以帮助改善桥本甲状腺炎的症状和病情。
Outlines
🌟 自身免疫疾病:未被察觉的流行病
心血管疾病和癌症分别是排名第一和第二的健康问题,但自身免疫疾病的患病人数超过了这两者的总和。Dr. Demitri 提到自身免疫疾病是慢性病的主要原因,尤其是Hashimoto病,这是一种自身免疫性甲状腺疾病。美国有2600万人患有Hashimoto病,绝大多数是女性。
🔍 Hashimoto病:不仅仅是甲状腺问题
Hashimoto病是导致甲状腺功能低下的主要原因,但它其实是免疫系统的问题。免疫系统攻击甲状腺,导致慢性炎症和器官破坏。治疗的目标是减少免疫系统的攻击,阻止炎症,改善甲状腺功能。
💡 Hashimoto病的症状和原因
低能量、体重调节困难和情绪不稳定是Hashimoto病的常见症状。女性患者由于激素周期的变化更易患病。患者常缺乏维生素D、胰岛素抵抗和多种微量营养素不足,这些因素共同导致线粒体功能障碍和能量水平下降。
📉 线粒体功能障碍与营养缺乏
线粒体是细胞的能量工厂,营养缺乏会导致其功能障碍。Hashimoto病患者通常存在多种微量营养素不足,如锌、镁和Omega-3等。这些不足导致线粒体效率下降,进而影响甲状腺的正常功能。
🔬 代谢组学测试:个性化诊断新方法
Dr. Demitri 采用代谢组学测试,通过测量代谢物的水平来评估患者的营养状态。这种测试能够检测出血液中未反映的营养需求,提供更全面和个性化的健康评估。
Mindmap
Keywords
💡自身免疫疾病
💡Hashimoto病
💡甲状腺功能减退
💡慢性炎症
💡自身抗体
💡维生素D缺乏
💡胰岛素抵抗
💡微量营养素缺乏
💡线粒体功能障碍
💡代谢组学
💡个性化治疗
Highlights
心血管病是排名第一的健康问题,癌症第二,但自身免疫疾病比两者加起来还要多。
自身免疫疾病是慢性疾病的首要原因,在美国约有5000万人受到影响。
最常见的自身免疫疾病是桥本病,这是一种甲状腺的自身免疫疾病。
大多数甲状腺功能减退症是由桥本病引起的。
桥本甲状腺炎是一种自身免疫性疾病,由免疫系统攻击甲状腺。
在美国,大约有2600万人患有桥本甲状腺炎,其中90%是女性。
甲状腺功能减退症的治疗不仅是补充甲状腺激素,还应关注免疫系统的异常。
甲状腺自身抗体的产生是免疫系统攻击甲状腺细胞的标志。
桥本病的诊断通常不通过活检,而是通过血液测试和超声波等方法。
桥本病的治疗目标是降低自身抗体水平,停止免疫系统对甲状腺的破坏。
甲状腺细胞的再生非常缓慢,需要七年时间。
甲状腺激素几乎影响身体的每一个细胞,直接作用于新陈代谢。
桥本病患者常见的症状包括能量低下、体重调节困难、激素失衡等。
桥本病患者100%存在维生素D缺乏,以及超过90%存在胰岛素抵抗问题。
患者常存在多种微量元素的边际缺乏,这些缺乏会干扰线粒体功能。
线粒体功能障碍是导致桥本病发展的基本问题之一。
医生使用代谢组学测试来检测身体内小分子代谢物的变化,以评估营养状态。
代谢组学测试提供了一种个性化的评估方法,与传统的营养素血液测试不同。
Transcripts
you know what's really fascinating about
the statistics on what type of health
conditions people get like
cardiovascular is number one cancer is
number two but there's some fascinating
information to show that there's another
epidemic condition that's even higher
than both of those combined and that is
autoimmune diseases and so today I
brought in a very competent doctor Dr
Demitri to discuss this this topic
because he has a lot of experience in
there so welcome Dr
Dimitri hello Eric hello Dr bur how are
you I I appreciate you taking the time I
I interviewed you before because you're
actually my doctor so I wanted to tap
into your knowledge base but you you
even mentioned this and this is like
mind-blowing there's so many people that
have autoimmune diseases nowadays um I
mean it's like it dorfs some of these
other conditions which is crazy but what
I want to uh ask you is out of all of
the autoimmune conditions that people
are getting what is at the top of the
list the number one
condition okay let me give a picture can
I show a picture of some stats that
could help uh um okay uh autoimmune
disease is the number one cause of
chronic disease and now I'm going to
refer a few statistics about us
population so while in US there are 9
million people suffering from cancer and
22 from cardiovascular disease there are
50 million probably more people
suffering from a type of autoimmune
diseases autoimmune diseases are
conditions where the immune system does
not recognize its tissues or organs
ourselves so it attacks them considers
them as being estrous uh U particles or
tissues and attacks them this creates a
chronic inflammation and gradually uh
leads to the destruction of the organ
interested now from all these diseases
the number one the most common one is
Hashimoto disease which is an
autoimmune thyroid autoimmune condition
yeah this is interesting because um you
know if you take a look at
hypothyroidism which you know you know
you just assume that the thyroid is not
working that well it's a thyroid problem
but isn't the majority of
hypothyroidism
hashimotos yes the great majority it's
the number one cause of hypothroidism is
Hashimoto's disease so in reality um
would you say that um this Hashimoto is
not necessarily a thyroid disease it's
more of an immune disease would I just
clarify that with me of course Hashimoto
thyroiditis is a autoimmune Condition
it's a chronic inflammatory condition of
the thyroid that is caused from the
immune system itself the immune system
attacks the thyroid gland and gradually
destroy it when the destruction of the
gland is above a certain percentage and
influences the function of the organ
itself we go into hypothroidism a lower
function of the thyroid gland at this
moment in us it's calculated that 26
million
of the population are suffering from
Hashimoto thyroiditis if you consider
that nine out of 10 of these patients
are
women it's a it's a huge part of the
women
population uh that is suffering from
this disease and it's pretty wild that
you you you know I know when I was in
practice people would come in with um
hypothyroidism and they would just think
oh I have low thyroid hormones so I just
need to take my thyroid hormones and
it's going to correct the problem but
this is a little
different this is a little bit
different the the problem here is that
since the thyroid gland is not
recognized from the immune system it uh
produces antibodies it they are called
Auto antibodies which attack the thyroid
gland and then these antibodies um get
attached to thyroid cells and then uh
cells of the immune system are guided
towards these antibodies and destroy the
gland itself so we have a chronic
inflammation of the thyroid but in order
to arrive at that point where the body
produces these antibodies and
distruction and chronic inflammation of
the gland there must be some prior
metabolic um dysfunctions and
deficiencies that brought to that
so uh it's a small this diagnosis it's a
small uh part of the whole problem the
whole
picture so basically there's a Cascade
of things and this is usually late on
the chain of events te tell me some of
the um potential triggers or
causes that precede this condition yes
um first of all while we said that there
are 26 million uh people with hashimotos
and nine out of 10 of these uh people
are women nine of
10 that are diagnosed with Hashimoto
thyroids are women this is caused mainly
because women have a lot of hormonal
adaptations and changes during their
lifetime they start having menstruation
they have mensturation every month they
get pregnant they stop menstruating and
so on and all these change in and out
and changes in the balance to
Progesterone and estrogen um create
adaptations of the immune system and the
theid has to adapt also and these
changes when there are uh the body is
not in an optimal condition guide to
dysfunction of the immune system and the
inflammation that causes the the disease
wow so 18% of the US population us
adults uh which is more than 60
million are have is calculated that they
have Auto antibodies against the
thyroid the Hashimoto is not one the AO
anti against the disease uh are one part
of the diagnosis in order to make the
diagnosis of Hashimoto thyroid it is the
actual is with
biopsy but usually we don't make biopsy
to everyone uh so we measure the TSA
which is a hormone secrete to regulate
pyoid function we measure the thyroid
hormones like T3 and T4 we also see
through
ultrasound uh test the form the the
structure of the theid and all these
data U contribute to make a
diagnosis but let's say that the final
diagnosis the is Hashim modus is a
biopsy diagnos noes about through about
so okay so then um now you you have a
you know a very large clinic in both
Rome and also Greece so what what are
you seeing
um as far as the common things to be
able to reverse this and first of all is
it reversible and then what what are you
finding that you have to change to make
things
better okay yes uh this is a very often
question we have if this is reversible
from patients also there are various
points we can intervene and this um uh
in relation to
reversibility uh when we ask about
reversibility we mean we stop the
destruction of the thyroid we mean the
inversion of the damage of the thyroid
or we mean stopping taking medications
because all this are different points
where the disease has arrived and uh uh
if we answer the first question uh so
stopping the immune system destroying
the thyroid gland and lowering the auto
adding bodies this is uh almost in 90%
of cases we can do that it it'ss um
usually in in six to months to a year
this is something that can um can occure
uh reversing the destruction of the thid
is probably the most important uh for me
and for the patients because uh we
Salvage the thyroid which is uh it's
vital for the quality of life and the
health of the patients and then uh
reversing also the
inflammation of the gland also this is
possible if it's in the um starting
stages of the disease but if the person
has started uh taking an um medication
and T4 usually uh to supplement the
thyroid with hormones uh this is not uh
in most of case this we cannot do uh
cannot reverse and stop taking the
medication because thyroid gland is a is
a gland that is very slow in
regenerating uh each tissue in our body
has a different time of uh regeneration
for example our blood cells regenerate
in a few months three four months uh the
skin cells in a few days and so on but
the theid cells regenerate
uh in they have a time of regeneration
of seven years so it's a very slow
process we had some cases that we follow
them for many years and it happens that
they had they managed to stop taking the
medication but it's not something we can
say for every case or this is going to
happen so reversing the disease means uh
initially the most the first Target
should be to lower the out antibodies so
this means that that the body stops
destroying the gland uh it means also
managing and stopping the inflammation
at the level of thyroid so the cells
that have remained uh they can function
in the best possible ways so this can
bring gradually to a diminishing of the
dose of the supplementation of the
hormone uh through a medication but
stopping complet completely the
medication once this uh the treatment
has started it's not something that we
can see that can it's possible for
everybody and as far as um you know we
know the thyroid hormones go into almost
every actually probably every single
cell in the body so they can they
directly affect the metabolism and so
many different things um what are some
of the common symptoms that someone gets
with a hypothyroid hashimotos type
condition the first symptom that
manifest is um and it's one of the Prime
symptom it's low
energy so the person doesn't have the
energy it used to have uh in Prior years
usually the people often think that this
is due to because they get older which
is not true uh it's a mitochondrial
dysfunction the the energy of the body
is lower um and I need to clarify this
point that the thyroid is a gland is a
it's a hormone that actually regulates
metabolism regulates the rate of
metabolism so our body doesn't function
uh in the same condition uh actually
these conditions change
continuously we are in a in indoors and
it's hot we go outside and it's cold we
can be angry we can be relaxed we can
eat we can
uh do physical exercise all these
conditions
uh need a different metabolic rate so
our body has to adapt to the needs of
energy and this the major adaptation
mechanism of the of this
um uh metabolic rates that change is
regulated through the thyroid gland so
thyroid increases the rate of metabolism
of decreases based on the needs when we
sleep we need lower energy and makes
that possible for the body to have the
Optimum output of energy so when the
thyroid
dysfunctions uh this means that our body
cannot adapt and this when we we speak
about adapting means about the function
of every organ almost every cell in the
body is influenced by the thyroid
hormones so by losing this ability to
adapt and uh respond to Chang in the
environment influences our uh health and
the function of the body as a a whole
one of the first things so that people
note is the lower of the energy levels
because the body cannot adapt to that
then uh the second is difficulty in
modulating regulating the body weight
people that usually have problems with
weight problems increased weight
sometimes even um they cannot get uh
increase their weight uh because have a
higher metabolic rate than needed uh
from the dysfunction of the thyroid but
the most common thing is that people
cannot adapt cannot uh regulate their
body weight they are overweight they
have more weight than they should
because the metabolic rate is low uh and
the body stores fat instead of being
able to to burn it and use it as energy
uh one one other thing is hormonal
hormonal imbalances so in women that
also most of the patients that suffer
from the disease are women we have uh
disregulated uh hormonal Cycles we have
um pain we have inflammation we have
infertility so many problems uh
migraines correlated uh with the
hormonal cycle and hormonal
imbalances another simp symptom which
also influen a lot the quality of life
of these patients is emotional
instability uh this people hasmot can
experience uh within the same day um a
lot of changes at emotional level they
can be tense they can be exhausted they
can be angry they can cry they have so
many Chang they often refer that they
are overwhelmed from thoughts that come
that they cannot control
and uh often they try to handle that
with uh meditation or other
means believing that this are due to
mental causes but the think it's a
hormonal and metabolic imbalance that
when this is corrected all these uh
problems uh uh get improve a lot if not
handled uh completely that's fantastic
what what are um as far as things that
you found are there multiple different
uh things that you're
finding that are like certain patients
are missing in their nutrition or things
or or have you found like there's like
only one thing that always causes this
problem or is it multiple
things actually there are uh some
metabolic dysfunction that are common to
most of the patients with hash Moto
with some
variations uh as in regard to deficiency
everybody I have not find the PA with
Hashimoto have vitamin D
deficiency so this is have vitamin D
deficiencies yes they they every
patients at the moment they are
diagnosed with hashimotos thyroiditis
they have vitamin D deficiency this is
100% or not close it's 100% we have made
the statistics we have announced the
results and it's 100% of patient with
Hashimoto have um vitamin D
defici then one common uh problem uh
also goes hand in hand with hash Moto is
insulin resistance uh also in this case
everybody with hash Moto or more than
90% in this case they are insulin
resistance or have insulin metabolic
dysfunction insulin their cells do not
respond uh to insulin signal they are
they have lost to degree the sensitivity
uh to
insulin uh another very vital problem is
micro multiple marginal micronutrient
deficiencies so these marginal
deficiencies along
with um insulin resistance also
disbiosis alteration of the
microbiome um they end to disrup
mitochondrial function so just to
clarify the marginal you're talking
about there's a um I guess subclinical
or slight deficiency of micronutrients
like trace minerals is that or is there
some other meaning of that okay when we
have a complete deficiency of vitamin
like vitamin D or vitamin C we have
brism have a complete a manifestation of
the disease that is completely uh caused
by the lack of this nutrient when we
have lack of vitamin C have scorbut uh
so in this cases we don't live in a an
environment that there is a complete
deficiency of a nutrient but we have
multiple marginal not complete but uh
deficiencies of micronutrients so they
lack of zinc
magnesium omega-3 and other minerals
potassium and so on uh chromium uh B
complex vitamins
uh vitamin E antioxidants amino acids uh
so the body in order to function needs
166 uh nutrients and elements and this
they have to be present all of them at
the same time in a correct equilibrium
and this can vary from person to person
uh in order for the body to function
optimally and these in most cases there
are multiple deficiencies this not one
of these various micronutrients that are
necessary for life to function to uh to
so our body can function
healthily the thing is that all these
multiple def uh deficiencies end up to
disrupt the mitochondrial function so
one
common uh problem here for this patient
probably one of the basic problems that
bring to the manifestation to the
development of Hashimoto theotis is
mitochondrial dysfunction mitochondria
are little organal that are within the
the cells that 90% of the energy in the
body is produced in these organal so all
these multiple deficiency even though
not complete deficiencies they disrupt
the function of the of the mitochondria
lower energy levels the efficiency of
the body and then the thyroid has to
work more m
so I got it it's a backup let's say that
your body has to produce 100% of energy
and your mitochondria function at the
50% level that's fascinating because so
in this
situation far is just overcompensating
for a lack of mitochondrial production
so one thing that's very unique about
what you do versus other doctors you
have a specialized test that you do that
really pick up um um these subtle
changes that really are very very
important almost like you get more of a
complete picture and I guess that would
be called metabolomics right yes true we
measure small molecules called
metabolites that participate in the
chemical reactions within our uh within
the human body so instead of measuring
the nutrients in the blood because this
does not reflect the deficiency or the
needs of our body because our body tries
to to keep the the concentration the
consistency of the blood uh constant
because if for example someone is
missing calcium even though he has
osteoporosis the levels of calcium in
the blood are going to be within normal
it's even in advanced stages of
osteoporo and lack of calcium because if
the concentrations of calcium go higher
or lower the heart will stop and many
functions or cells will die so the
body tries uh to keep the consistency of
the blood stable so when the
consistency the the levels of a nutrient
the blood changes this means it's very
late it's too too late in order to
overcome this problem we have to measure
chemical reactions that Cur the presence
of the nutrient we want to test for
example we can measure uh the uh
transformation of one metabolite to
another that needs B12 or needs
magnesium on its glutamine and so on uh
so by measuring this chemical reaction
if this is occuring at a normal levels
optimal levels we can see if uh the body
needs more or less of the nutrient we
are testing so basically you know
instead of looking at the uh rdas for
certain nutrients we're looking at how
these rdas are really being utilized in
the body and seeing if they're
sufficient or not depending on that
individual so it's a
uh uh completely different strategy yes
it's individualized yes personalized so
so for those of you listening if you if
you have a condition like this and you
want to get it evaluated a lot deeper
I'm going to put your link down below
where you can um get this test get it
evaluated um I will put that link down
below and I know that you mentioned that
you you have a small amount of time to
do this interview so I just wanted to
cram as much as I can in this interview
and
and just get these uh the overview of
the thyroid and this is just fascinating
information so anyway I want to thank
you for spending the time and uh
hopefully clarifying some of these
confusions that people have about this
thyroid problem hashimotos and not to
mention just autoimmune in general and
how different that is so thank you so
much thank you thank you Dr B for the
interview you're welcome
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